Hard Truths & Convenient Lies

Tin Foil, the NSA and Dr Jayadevan. Original Antigenic Sin and Vaccines.

June 07, 2023 Steven A.R. Murphy MD
Tin Foil, the NSA and Dr Jayadevan. Original Antigenic Sin and Vaccines.
Hard Truths & Convenient Lies
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Hard Truths & Convenient Lies
Tin Foil, the NSA and Dr Jayadevan. Original Antigenic Sin and Vaccines.
Jun 07, 2023
Steven A.R. Murphy MD

In this episode, Dr. Rajeev Jayadevan, Co-Chairman of the National IMA COVID Task Force, along with co-hosts Alex and Dr. Murphy, explore the concept of vaccine imprinting and its relevance to our response to new COVID-19 variants. They explore a recent study that compared the antibody responses of individuals who received different types of COVID-19 vaccines. The study suggests that previous vaccinations may impact how our bodies respond to future variants. Stay informed and discover the evolving landscape of vaccine research in this informative discussion.

Show Notes Transcript

In this episode, Dr. Rajeev Jayadevan, Co-Chairman of the National IMA COVID Task Force, along with co-hosts Alex and Dr. Murphy, explore the concept of vaccine imprinting and its relevance to our response to new COVID-19 variants. They explore a recent study that compared the antibody responses of individuals who received different types of COVID-19 vaccines. The study suggests that previous vaccinations may impact how our bodies respond to future variants. Stay informed and discover the evolving landscape of vaccine research in this informative discussion.





  1. Hard Truths and Convenient Lie...jeev
  2. Jayadevan) mastered audio
  3. Wed, May 17, 2023 12:30PM • 54:02
  4. SUMMARY KEYWORDS
  5. people, vaccine, antibodies, virus, alex, death, rajiv, study,
  6. talking, micron, infected, mrna, vaccinated,
  7. mask, dose, t cells, barcode, infections, diabetes, outcome
  8. 00:09
  9. hard truths and convenient lies is a podcast dedicated to lifting the veil and exposing the reality of our
  10. dysfunctional government, economy and
  11. healthcare system. The opinions today expressed by Dr.
  12. Stephen Murphy and entrepreneur Alex Michigan addressed that nothing we say today should be
  13. considered medical, legal, financial, or tax advice. Go get your professional for that. We have potty
  14. mouth,
  15. and we're here to lift the veil and expose the hard truth and convenient eyes
  16. 00:42
  17. oh man, Alex, Another week, another week of getting fat on convenient lies. This is crazy. So with all
  18. the madness that has happened, how was your week?
  19. 00:57
  20. I mean, ho
  21. w can you not celebrate pandemics officially over this week? We did it. No more COVID
  22. Then we're long COVID It's over.
  23. 01:08
  24. It's over. It's the shortest case alone COVID you'll ever have.
  25. 01:11
  26. Yeah, we were just chatting offline about you know,
  27. solutions, the long Cove and how you can make it
  28. short. COVID ya know? You know, Canada has a lot of good solutions with euthanasia.
  29. 01:24
  30. They got a program up there with JT. I think he's got he's got the fast track to long COVID. And so
  31. today, Alex, for
  32. you. I brought on an amazing guest. We are really excited today because he is going to
  33. literally give us the hard truth about by Vaillant vaccines and COVID. All right, so today, we need to
  34. buckle up because it's gonna be a bumpy one. So be prepared. We a
  35. re here today with Dr. Rajiv Jaya
  36. David, who's not just a doctor and a specialist with extensive experience in gastroenterology. But he is
  37. a smart one, he earned top honors and both his MBBS and his MD over in valore India. And in addition
  38. to that, he's go
  39. t clinical training in epidemiology and public health from the Netherlands. So I don't think
  40. we're going to have more of an expert and I know how you feel about experts, Alex, where we're gonna
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  45. https://otter.ai
  46. cut to the quick today, we're gonna identify the experience he
  47. re, and we're going to talk about a recent
  48. study link that he put out in regards to buy violent COVID
  49. -
  50. 19 mRNA vaccines, so we're gonna cut to it
  51. today. So Dr. Dr. Jaya, David, we got a little mic issue going on with Rajiv and that's okay. So Alex, you
  52. have
  53. met Rajiv before, right?
  54. 02:49
  55. So one of my key influences over the years on COVID, you know, I see him as a one of those experts
  56. that is telling the hard truths, not afraid to go against the grain, and really share important information,
  57. fact based info
  58. rmation, just like his information that he's going to share on the latest vaccine. And it's
  59. true efficacy around that. So, you know, the interesting thing is, is you know, Rajiv has been a source of
  60. really great information, not only in what's going on in
  61. India, right, but really around the globe here. And
  62. as we know, we have different vaccines that have been used. And I think what's really important and
  63. the information he's going to share is what the Western world felt was going to be, again, the
  64. 03:40
  65. so
  66. lution to solving the pandemic. Love it, that you imagined it wasn't the solution.
  67. 03:48
  68. Alex I'm so obese with convenient lies with these guys. I don't know what else to say. You know,
  69. 03:57
  70. obviously, this morning, I
  71. 03:58
  72. was so excited when they sai
  73. d they were indicting somebody for lying to Congress.
  74. 04:04
  75. Even better. Even better, it was a congressman. So we were like, Oh, this could get great here.
  76. 04:11
  77. Wrong. It's the Santos right. The Santos clause. So with the thing was Rajiv that we had
  78. 04
  79. :20
  80. he told me that the government is turning off his mic and shutting him
  81. 04:23
  82. up. I think that was what's going on. Right. Your next because you talked about Santos. Alex, I don't
  83. know what to say here. Other than the fact that what we have going on is
  84. just despicable. And with I
  85. don't know, are you have you been playing around with Chet GPT and auto GPT
  86. 04:44
  87. Oh, my God. Yeah, we're obsessed with. That's all we're working
  88. 04:47
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  94. on is working with arms,
  95. 04:48
  96. but I want you to think of something. So
  97. they left this out to the public now. This obviously is something
  98. that the CIA
  99. 04:58
  100. had it was your tinfoil there. Oh, Alex, they've had these really? Okay, listen, all right,
  101. 05:04
  102. there's no doubt that the NSA has some of the smartest human beings in
  103. the world working for it. That's
  104. one of the misnomers, like, Okay, I'm a big, you know, opinionated person that government employees
  105. are not necessarily federal government's filled up with a lot of fat, let's just put that right. But the NSA is
  106. got some br
  107. illiant, brilliant people. And the things that we're able to do from a technology standpoint,
  108. will make you blush inside the government compared to Google and Apple and Amazon, this amazing
  109. technologist. So you know, things like MLMs, ya know, they're not
  110. new to the intelligence agency. But
  111. open AI, you know, that is a private initiative. You know, it is a it is a technology that is extremely
  112. exciting, even though as we're seeing with Facebook, open sourcing and everything else. It's not as
  113. novel from pure
  114. AI as some people thought. But what is novel is around how they're assembling a
  115. platform, plugins, and so forth, and use the auto GPT. and things of that nature, where I strongly
  116. strongly believe that software stacks top the bottom, small companies, big co
  117. mpanies across every
  118. industry will be rewritten. I mean, we are doing it. I mean, we are literally I pulled the plug on four or
  119. five, what I call legacy projects. It's just fucking stop. Because this is the future this is everything is
  120. going to be rebuilt,
  121. just like things were rebuilt around the iPhone. And you know, there was a time of
  122. rebuilding around social graph and all these kinds of things. But this is a moment that will change the
  123. entire world. And it's not going to be just open a eyes LM, it's goi
  124. ng to be the idea that this is the
  125. beginning face. Five years from now, we're probably going to see another massive moment of AI
  126. models.
  127. 07:08
  128. But why so long? Why so far out,
  129. 07:10
  130. that's about how long it takes, if you think about it for these major te
  131. chnology revolutions. And the thing
  132. that's still missing from the LLM is the ability to really make decisions. So decision making or actually,
  133. you know, rational decision making her fourth James, are you there? Yes, I'm missing me,
  134. 07:32
  135. right. No, no, th
  136. is is good. We get it. We'll talk about AI with Rajiv too.
  137. 07:36
  138. So we've got super smart guy, Rajiv here. We've got super smart guy, Alex here, and we got just a
  139. doctor and Murphy and we are here today, to take that play to convenient lies away. Nothing
  140. but a
  141. spoon full of truth from Rajiv. So Rajiv, as I got through it before I was talking about your experience,
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  147. not just in India, and the United States and, and Ireland. Gotta love it with the Murphy. And you also
  148. served as the CO chairman of the National
  149. ima COVID Task Force, and you're past president of
  150. 08:11
  151. Murphy. Why did we cover this?
  152. 08:14
  153. I'm fake Irish. I'm actually adopted Irish.
  154. 08:17
  155. I'm Puerto Rican.
  156. 08:21
  157. Bullshit, you know,
  158. 08:23
  159. it's still an island. It's still an island. Come on.
  160. It's a little warmer down there.
  161. 08:28
  162. You know, Congress talking about long COVID.
  163. 08:32
  164. Island up like Santos.
  165. 08:35
  166. Alright, so we're Jeep seriously, we have to talk about a lot of stuff. But I think the first thing that we
  167. have to talk about with yo
  168. u, we got to get over this first and then we'll go through the rest of it. So bear
  169. with us, because we just got to know you just dropped a bombshell onto Bio Archive. Can you talk to us
  170. a little bit about your study that where you investigated COVID
  171. -
  172. 19 mRN
  173. A vaccines? And tell us about
  174. that? Tell us what the heck you found what's going on?
  175. 09:06
  176. Okay, so thanks for having me on the show. Pleasure to be here. And greetings to all our listeners.
  177. 09:13
  178. Yeah, I know. You've said that the beginning. We'll see
  179. if you got pleasure at the back end.
  180. 09:20
  181. Yeah, so the discussion is on a preprint that came on a preprint server, I believe it's mid archive, on a
  182. on a study on a 72 individuals who had different types of, let's say, vaccine combinations of the mRNA
  183. and
  184. genre, right. So here's here if you people reading from my notes, they all got three doses of the old
  185. vaccine with
  186. 09:53
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  192. the old them or
  193. 09:55
  194. the old mRNA vaccine.
  195. 09:57
  196. So what is subway punchcard that said the More you get the you get a free sub.
  197. 1
  198. 0:02
  199. Oh yeah, right. Yeah, the more the merrier. Yeah. And then they were divided into two groups. And
  200. then one group, one sub group got the old vaccine as a fourth dose. And then the other got the new
  201. one. That's the Bible and vaccine. And as our
  202. listeners will understand the Bible, it has two spikes on it.
  203. That is the spike of the Omicron and the spike of the old virus. So, so we have, so we basically have
  204. comparative visually comparing two groups of people, right, with two combinations of vaccina
  205. tions. And
  206. then some of them underwent what is called Breakthrough infections, which is in plain English.
  207. infections that occur despite vaccination mean, the vaccine didn't work. It did not work doing not
  208. working for that. Now, it didn't prevent In fact,
  209. 10:57
  210. I was told that if you
  211. 10:59
  212. vaccinate you don't carry the virus.
  213. 11:02
  214. Yeah, you don't have to add a mask. Right. Take your mask off. If your vaccination there was a time
  215. Yeah, so
  216. 11:10
  217. yeah, she's gone. By the way, she's gone. So see you later.
  218. Bye, Rochelle.
  219. 11:17
  220. Alright, so as the study as they looked at these participants in the in the study, they found very little
  221. difference between the old and the new vaccine. So that's, that's the long and short of
  222. 11:28
  223. it. Right? So these were they
  224. wouldn't mean they found that you mean, they weren't different at all?
  225. 11:34
  226. They ran, they ran in, in any sense. They were they were no different. So let's see, let's see what they
  227. could have measure. Now, what would you expect a vaccine to do a, it woul
  228. d, you would expect it to
  229. work on your immune system, and your immune system will go to work after it gets a signal and it
  230. makes some antibodies for you. And then some other components that we will not be talking about in
  231. detail will stick to the antibody
  232. section here of our immune response. So you would expect the vaccine
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  238. to generate antibody out anti bodies have the right kind, particularly that could cover the latest versions
  239. of the virus? That's really the objective of getting an updated vaccine, isn't
  240. it? But unfortunately, when
  241. they compared the two groups, they found that the old vaccine worked just as well or the same as the
  242. new vaccine. So why would that be? Why would a new a graded vaccine with an O micron spike on it?
  243. Why would it not generate an
  244. A microns specific antibody response? That's a logical question that
  245. would come on our minds would come on anyone's mind because after all, you're getting an upgraded
  246. vaccine. So the answer to that is, when they looked at the antibody types, the compared b
  247. etween the
  248. old and the new. They found that even when a new vaccine was given the body remembered the old
  249. vaccine, it's like it's the term for that is called immune imprinting, or colloquially, it used to be called the
  250. original antigenic sin now that that'
  251. s a rather dramatic term which is inaccurate and has been
  252. discarded, but I love that because it says a lot. So is the original sin. Right? Has that. That that very
  253. mystical feel to it? I don't know.
  254. 13:31
  255. It sounds like Madonna to me, I feel like about Ma
  256. donna.
  257. 13:35
  258. Okay, so it could be beautiful stranger. That's my favorite track of nice. Now, let's look at what Original
  259. Sin is. And for for our listeners again, let's let's just take an analogy of ice cream. Let's say the first ice
  260. cream we ever had was
  261. chocolate. The next time you have let's say strawberry flavor. If you continue to
  262. believe that you haven't chocolate, and then you have a problem, and that is called the original
  263. antigenic sin. They have logically your mind is or someone else is telling yo
  264. u, hey, I'm giving you a
  265. strawberry. But you might say no, this is chocolate because chocolate is what I remember. So likewise,
  266. our original vaccines all over the world, whether it's mRNA or adenovirus vector or subunit vaccine or
  267. inactivated, they were al
  268. l built on the ancient version of the virus. So they had our immune systems
  269. programmed with all these beautiful memory cells that would perhaps live forever in our in our tissues.
  270. For example, in our bone marrow they have they have unlimited longevity, hop
  271. efully. And when the new
  272. let's say when the new antigen comes in, now antigen current can come in in two forms either as a
  273. vaccine or as infection. Now let's say you take a shot of the new vaccine, our immune system still
  274. remembers the old memory. So it it
  275. evokes the old memories and the old antibodies get generated.
  276. Now, these post antibodies are not so bad themselves they are, they can cross cover quite a bit. And
  277. it's not just the antibodies that make our immune response. We've got plenty of other compon
  278. ents of
  279. immune response like the T cells, you know, everyone knows what T cells I think thanks to COVID now
  280. 15:29
  281. Rajeev, we have a long talk about that with Dr. Leonardi. So um, for the listeners, you want to listen to
  282. episode, a couple episodes go where
  283. you want to listen to him, Anthony, Leonardo, he talked a lot
  284. about T cells,
  285. 15:43
  286. right. So these T cells, they are not particularly fooled by the newer versions of their micron so much as
  287. antibodies are. Now I'll explain how that works. You know, for o
  288. ur for our listeners, let's say let's
  289. imagine that antibodies and T cells are to, let's say two types of immune response. And they and their
  290. job is to recognize the enemy. And you could recognize the enemy by using a barcode, let's say you go
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  296. to the superm
  297. arket, you pick up a bottle of shampoo, and then you give it to the to the clerk at the
  298. supermarket counter. And they scan it with and scan the barcode. And it's a barcode that tells the clerk
  299. and the computer that it's a bottle of shampoo. Now with the ba
  300. rcode is changed, then the computer
  301. shows and era it doesn't recognize the product. So when the virus changes its genetic structure, the
  302. barcode also changes. Now the change in the barcode affects the antibody section more than the T
  303. cells. So the T cells
  304. are not so special specific for these barcodes, they work on a different set of
  305. barcodes, if you will, their barcodes are smaller, whereas the antibodies tend to work on very large
  306. barcodes, which means that it's easier to change a little bit of descriptio
  307. n or a little bit of the code on the
  308. barcodes, which is why antibodies are fooled quicker than your T cells. So back to our study over here
  309. the study that was published that was that is uploaded on preprint. I did not do the study, I did the
  310. analysis on it
  311. right on Twitter. That study shows that your antibodies are not particularly smarter. That
  312. study does not really look at T cells, T cells are difficult to measure the cumbersome and you really
  313. cannot estimate how good they are in terms of clinical outcome
  314. s. So in other words, you may find a T
  315. cell hanging around here and there. But you can't tell if that T cell is going to do something for you.
  316. Whereas antibodies, on the other hand are more readily measurable. And there are studies that show
  317. that antibodie
  318. s correlate to some degree with clinical outcomes. For instance, there are some more
  319. recent paper that came out that that looked at a fantastic study that followed people over a period of
  320. time and looked at the antibody levels and try to see if these peopl
  321. e are getting infected. And they really
  322. found that those who had high antibody levels that is neutralizing type. Were not getting infected so
  323. much as the other people. So there we are. So back to our study, whether you took the Bible and or the
  324. monocular,
  325. and the antibody response was practically the same over to you.
  326. 18:27
  327. Okay, so question here to peel back this onion, original allergy and concept. In this study, did it break
  328. down individuals that had different amounts of doses so that they were original
  329. ly boosted with the
  330. original vaccine? Maybe individuals that had two mRNA versus one had no virus, vector virus, danger,
  331. a type of vaccine, they break that down at all, even someone who was partially vaccinated. One mRNA
  332. shot was any of that in the study a
  333. t all.
  334. 19:07
  335. You know, I have to check that again. If you weren't, I can, I can look it up right now. But my
  336. understanding is that they got three doses of Manorville. And at baseline. So these were that was a
  337. baseline
  338. 19:18
  339. booster. So basically, we're l
  340. ooking at individuals. So you're talking about vaccine, you know, maximize
  341. the individuals, right? Yes, that are lined up for the latest thing bring Yes, correct, you know, Fauci
  342. shots.
  343. 19:33
  344. I told you the subway card, did you need one more and they get
  345. that footlong Italian combo.
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  352. So, you know, from a science standpoint, Rajiv, do you think there's potentially any merit to those that
  353. got many vaccines, having, you know, a larger hurdle from a antibody standpoint of developing you
  354. know, antibodies
  355. to A newer strain of COVID, whether it's developed through a vaccine, or through an
  356. infection,
  357. 20:07
  358. yes. Now the answer to this question is directly in this study, they found that people who had
  359. breakthrough infections generated antibody antibodies that
  360. were more relevant to the purpose as
  361. opposed to people who received the vaccine. So which means that as the as you, as you follow a group
  362. of people over a timeline, as they get infected over and over, with newer and newer versions of micron,
  363. the body is ab
  364. le to generate antibodies to the newer versions. So that is something that the study
  365. discovered, but you can't prepare them in advance. So in other words, you can dissipate a new variant
  366. coming down, say, let's say six months from now and prepare the body
  367. ahead of time using this Bible
  368. and vaccine. That was my hope as well. But that did not. Yeah.
  369. 21:05
  370. I'm actually
  371. 21:06
  372. hypothesizing something a little bit different. And this is been one of my concerns. And I'm afraid that
  373. there hasn't been studies beca
  374. use you don't study what you don't want to know, as someone at the
  375. CDC told me over two years ago, about breakthrough infections, you don't track what you don't want to
  376. know, quote, unquote, government official. Um, but, you know, the question I have is so
  377. meone who is
  378. well primed, three mRNA shots for the wild type, let's say it was never infected, which is, I think, a large
  379. percentage of the American population. And then they go out, they get infected, they get boosted, and
  380. probably continues that nice lit
  381. tle cycle versus a population that was either infected with the wild type or
  382. delta may or may not have been vaccinated there after, you know, comparing the difference of the
  383. antibody response to subsequent exposures to the virus, whether through vaccine or
  384. infection, you
  385. know, any idea of anything that you've read whether this study or another study, how antibody
  386. response may differ between those two?
  387. 22:36
  388. Now, let me let me simplify the question. For our viewers what Alex said was comparing people who
  389. re
  390. ceived multiple doses of vaccine to people who've been infected Is that is that the set summarize
  391. your question,
  392. 22:53
  393. who infected or a minimal amount of the vaccine one dose one more primary primary series?
  394. 23:01
  395. So what what has been? What is? What is
  396. clear is, if you look at the CDCs mortality graph, that is
  397. something I've been tracking over the years, they have beautiful graphs that have shown the truth all
  398. along, but they don't get much attention, except by a few people like yourself and me and a fe
  399. w others
  400. who who love to look at the graph in updated forms. Now, there are two ways you can look at the
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  406. graph. The graph, you got to go to CDC site, look at the mortality patterns by age group. Now, if you
  407. click on the right icon, you can actually divide
  408. the graph by age group. And that's important because we
  409. know that COVID affects different age groups differently by a vast amount. In other words, people who
  410. are over 6065 are at much greater risk compared to someone who is below let's say, 45, or even 30.
  411. So if you compare the mortality rates and death rates between age groups, you can see that below 50
  412. below the age of 50. There is very little difference between any subgroup whether they are infected with
  413. a vaccinated whether unvaccinated with a boosted w
  414. ith Manorville and violent to those three dose.
  415. They're all the same in terms of deaths. But when we look at people over 50 If you look at the CDC
  416. mortality data, you will see that people who have received any vaccine, let's say a baseline vaccination,
  417. let
  418. 's say two dose of mRNA with a three dose with a third dose as a booster what what they call a
  419. booster some people think the mRNA is actually a three dose vaccine. I believe that because the first
  420. two doses were given too close together that they actually
  421. worked. Too many people believe that they
  422. the first two doses, effectively what this is saying The dose, the third dose of the mRNA worked as a
  423. second dose. Alright, so let's if you look at that category, over the age of 50, there is a significant
  424. differen
  425. ce in death rate between people who have not received a vaccine and people who receive the
  426. vaccine. But if you look at people who receive more and more and more dose of vaccine, you hardly
  427. see any difference. Except in the 80 Plus segment, if you look at t
  428. he 80 Plus segment on CDC graph,
  429. you will see that people who've received the updated booster there is a difference in the death rate. So
  430. if there's one segment, I would single out to receive the Bible and vaccine as it's available, I would say
  431. it will ben
  432. efit someone over the age of 80, who's at greater risk? And if it perhaps not benefit at all,
  433. someone who's below 50
  434. 25:58
  435. Yeah, I mean, I think from what I've seen, and correct me if I'm wrong, either of you. I think the data is
  436. extremely strong, that
  437. all the vaccines help prevent or decrease death for a period of time after
  438. receiving a vaccine, whatever it is, they they are effective at reducing death in the elderly and immune
  439. compromised, and possibly across other demographics. For a short, short peri
  440. od of time, I've been, you
  441. know, beating the dead horse on that for years on, it's consistent with the flu vaccine 90 to 120. Um, but
  442. that's actually not what I'm thinking about. And maybe I'll be a trot out who tried to say this a different
  443. way. I'll ask
  444. it really controversial. Do you think that people that have received a lot of vaccines for the
  445. original wild type are at a disadvantage, the people that only received the primary series because their
  446. body is imprinted? More and more and more and more with
  447. a virus that is radically different than what's
  448. circulating right now? XP b dot whatever you want to wherever we are right now I've kind of lost track
  449. SBB
  450. 27:18
  451. I know and then you're going to add all those all those all those memes that makes trouble for
  452. me the
  453. acronym and then you got to the home of the ox. All these other words, which are like
  454. 27:33
  455. I mean, you have you have you know the wild pipe all the way over here. You have Omicron the Baa
  456. Baa strains, just call it and now you have XP so XP be cir
  457. culating are people that have been maximum
  458. you know, maximize their vaccinations, wild type advantage for developing public bodies under gets a
  459. SPV?
  460. -
  461. 10
  462. -
  463. Transcribed by
  464. https://otter.ai
  465. 28:06
  466. Hypothetically, hypothetically, yes. I'll refer you to a researcher whose name is Yong Yong Chol ca
  467. Oh,
  468. Richard is a middle name. And Richard Chou has published an extraordinary number of of studies on
  469. practically every variant that came along. And his his his his findings are spot on. So I will. I'm sure
  470. you've you've seen his work. I've routinely analy
  471. zed and tweeted his work. We follow each other. And
  472. the answer to your question, Alex, is in one of his recent works. You asked if people who've been hyper
  473. vaccinated could be at a disadvantage. I know there's no set stone. I'm just making that up, right.
  474. What
  475. Dr. Chow found is that the more number of shots you've taken off the original vaccine, the stronger your
  476. Imprinting is, that is your most stubborn your immune system is more stubborn. So it might not
  477. theoretically generate. It might not have the flexi
  478. bility to adapt to something new. That is a sense we got
  479. but here's the most exciting part. He also found that this could be overcome with repeated exposure to
  480. the antigen. For instance, someone who has had multiple or micron breakthrough infections. You s
  481. aw
  482. the CDC study from France that showed that you could get reinfected in two months. One out of seven
  483. reactions occur on two months and someone who goes to the club or to
  484. 29:53
  485. UCF. I mean, you see SF van right, then you should just go out and get your m
  486. oney and just keep on
  487. getting Omicron.
  488. 30:03
  489. Miami cubish live in Miami. Now, event that comes in Dr. Charles group showed that if you've had to
  490. exposures to the recent versions of flavors of employment, cron, yes, your immune system is able to
  491. overcome t
  492. he so called imprinting. So in other words, the ancient memory that it is, it seems to evoke
  493. each time as no longer a problem. And he also showed us in mice and in humans, that if you give two
  494. doses of the updated booster, you were able to generate a bette
  495. r response than with one dose. Now
  496. I'm not endorsing, giving multiple doses of a violent or updated vaccine. I'm giving you the observation
  497. here. Now, that brings us to the question, what are we trying to achieve here? That's a very important
  498. question. Wha
  499. t are we trying to achieve? Are we achieved only trying to achieve the highest number of
  500. shots per person? Or are we trying to achieve the best outcomes for the community now both seem to
  501. be a little divergent here, if you look at experience across the wor
  502. ld, look at South Africa, for example,
  503. one of the, let's say the moderately vaccinated countries, vaccination rates in South Africa are lower
  504. than that of India, where I live in now. They have not had a major surge of severe disease or deaths in
  505. the last y
  506. ear and a half. They've had a fair share of zero micron waves, they've had delta wave, they've
  507. had multiple other waves prior to that. And they've definitely not had a huge number of deaths, agreed
  508. that the documentation in South Africa and the documentati
  509. on India would not be as good or as diligent
  510. as that in the United States, or Germany or Japan. But that's that's a strong observation, then South
  511. Africa, which got some of the world's top scientists who keep publishing from there, they have also
  512. observed
  513. that their death rates are not so much. So it is not like, if you've taken more vaccines, the
  514. death rates go even low. That doesn't seem to be the case, to me at least. So that appears to me to
  515. answer your question very directly. I believe that if you prov
  516. ide adequate immune memory, which is
  517. long lasting, in the three and a half years of the pandemic, your chance of death is minimized for the
  518. age group you belong to, so to belong to an old age group,
  519. -
  520. 11
  521. -
  522. Transcribed by
  523. https://otter.ai
  524. 32:33
  525. then why are we still talking about COVID? Right.
  526. 32:35
  527. And we're not right. That's why we declared the pandemic over.
  528. 32:38
  529. I mean, but you know, the group of us are, so why Why should Why should anyone else care? Are we
  530. alarmists? I mean, is COVID. really mild? Is COVID Just the flu? Or is this just t
  531. he new respiratory virus
  532. of a generation that, you know, getting COVID Multiple times a year? Is okay, I mean, should should we
  533. all just like pack it up? And, you know, the volume and and deal with it?
  534. 33:07
  535. Good question. You know, I would I would say th
  536. is. Most people seem to enjoy the narrative that the
  537. COVID is a common cold. Why? Because it produces the symptoms of common cold. I mean, let me
  538. ask you this question. On the polio virus it gives you diarrhea, no, diarrheal disease? No, I wouldn't
  539. want po
  540. lio. It gives me diarrhea. Disease just take take Pedialyte take some take some MSN, or Dan
  541. cetera. No, that doesn't fix polio. Now, just because the symptoms you observed from COVID
  542. happened to be a runny nose. That doesn't mean that runny nose is all tha
  543. t this virus is causing this
  544. virus affects your circulatory system, the inner lining of your blood vessels now in the lining of our blood
  545. vessels called endothelial, this is not a piece of our paper. This is a living, complex living system. It's a
  546. whole bi
  547. ological system in itself. It controls your blood pressure, it controls your vascular turn, it controls
  548. atherosclerosis, it controls everything that delicate lining now, if you have a virus running around, that is
  549. affecting your endothelial every month, ev
  550. ery second month, you know what I would prefer not to I
  551. would not I would not want that virus in my circulatory system. Now that's if you have a problem in your
  552. circulatory system, you're not going to get symptoms you will not until something tends to be h
  553. appens
  554. for example, somebody gets a stroke or a heart attack. You have we excess death rates coming in. If
  555. you see the Australian Archer accurate data, you see a percent increase over baseline sips or micron.
  556. Hey, you got your excess that's coming in and t
  557. hat is one was likely the result of escalate events that
  558. are occurring down the line. Now, these are people who don't die from COVID. Right? So you're
  559. 35:07
  560. not going from so you're not going to see that in the data. Right? Right. Rajiv, you won't have to
  561. wait
  562. until the life insurance payout or until we've got an entire society that is so demented from stroke
  563. dementia, that it's like the movie Idiocracy, right? I mean, I mean,
  564. 35:25
  565. but I mean, just, you just look at any chronic disease in America or even
  566. the Western world, you look at
  567. diabetes. You know, it kind of blows my mind the the level of acceptance that people have, I mean,
  568. obviously a huge issue in India, you know, that it's, like a foregone conclusion that most people will just
  569. accept getting di
  570. abetes, they have pre diabetes, it's the on ramp, you know, I'm gonna go to Dr.
  571. Murphy. He's gonna give me my fancy little drugs from Novo Nordisk. You know, because they came
  572. over and gave him a lunch, and that's all it takes.
  573. -
  574. 12
  575. -
  576. Transcribed by
  577. https://otter.ai
  578. 36:06
  579. It's not all it takes
  580. , it's all we can get Alex, a different animal.
  581. 36:13
  582. To act is compliant. That's all we can get now. But, um, and it's the on ramp and you you get diabetes.
  583. So, you know, isn't long COVID Whether the degree of severity of it is, it's just human humankind
  584. going
  585. to accept a new chronic illness? That is part of modern society, because people value their lifestyle
  586. more than their health.
  587. 36:44
  588. Good question. So let me pick.
  589. 36:51
  590. Just fucking ask that, Alex. I love you. You're freaking certified genius. But
  591. what did you just ask you
  592. basically, you mean people are going to accept chronic disease? Because they like Big Macs and bug
  593. lights and cigarettes and sex with prostitutes? Like, come on.
  594. 37:10
  595. Face it. I mean, like, there's people like talk about monkey
  596. pox for a second, that, you know, we're
  597. talking about HIV and everything else. Yeah. And it's like, but don't you understand that these people
  598. just have sex without condoms. That's just the way of life in the community. They got HIV, of course,
  599. the gonna
  600. get monkey pox. That's the whole reason. And it's like attacking like people like us that are
  601. bringing up the risk. It's like with
  602. 37:33
  603. HIV, because they won't wear a condom. Lucky guy is gonna say, Well, we're kind of the vaccinated,
  604. but there's
  605. 37:41
  606. no shit, right? Hello.
  607. 37:43
  608. It's like, Wait a second. So Haven't we just turned every fucking disease into a massive circulating shit
  609. show if you're exposed to it? But yeah,
  610. 37:53
  611. dancer Alex is? Yes. Our art the hard truth, seriously the hard truth? Ou
  612. r government is going to
  613. encourage us to make a risk calculation. That being at home is just not worth it.
  614. 38:12
  615. Yeah, I mean, but it's not. I mean, you don't have to stay at home to avoid COVID.
  616. 38:15
  617. Oh, well, that I that's the narrative that's going
  618. -
  619. 13
  620. -
  621. Transcribed by
  622. https://otter.ai
  623. 38:19
  624. to be painted. I live a fairly normal life.
  625. 38:24
  626. risk adjusted normal for Los Angeles.
  627. 38:27
  628. I clarify that by May I mean, when Miami and wherever the heck I am, I was just in Europe. I wore my
  629. n95 Mask. Yeah, I'm, you know, I'm going through
  630. airports, I don't have my mask on outside, I'm not
  631. one of those people that you know, want to get labeled that Oh, my God, you know,
  632. 38:45
  633. I'm wearing a mask everywhere. No, I'm wearing a mask in high risk situations, I make decisions that
  634. minimize that l
  635. evel of exposure, I'm not gonna also go into a crowd of 5000 people with a mask on if I
  636. don't have to be there. If I have to get through an airport, I will try to get through there as fast as
  637. possible. And minimize exposure, especially in times where I fee
  638. l that it's higher risk or in an area that's
  639. higher risk. So I mean, you know, let's not go to the extreme of the silliness. The government wants to
  640. promote, but that's also like, No, I could go to McDonald's and eat at McDonald's and not get diabetes
  641. and
  642. become obese. You can it's it's level of exposure, right? It's control. It's not like Oh, give me all this
  643. virus. Or give me
  644. 39:37
  645. whatever Jim says. He says if you get the virus exposure a bunch of times you are going to be more
  646. antigenically adapt.
  647. 39:
  648. 45
  649. Well, that's the case specially for those that had all that extra vaccine that they didn't need. But I have
  650. no easy way where we it's kind of get back when No,
  651. 39:54
  652. we're really talking about the fact that are we going to have to live with this virus
  653. rich Yeah,
  654. 40:00
  655. I mean, I think I think the reality is what we're saying here. What, you know whether you're vaccinated
  656. or not vaccinated, tell me, Rajeev, I'm getting this wrong. If you're catching the virus, even if it appears
  657. to be mild, upper
  658. respiratory symptoms, you're causing damage to your vascular system. And that
  659. damage is completely unknown. What it means, you know, ignoring all of a sudden deaths by 30s and
  660. 40 year olds right now, that's unrelated of course. But, you know, it's truly un
  661. known what that is going to
  662. cause in the future.
  663. 40:39

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